A new study by Hunt et al. claims that BPA has negative effects on pregnant monkeys and could be a concern for humans. The study is actually a small scale study and the single oral dose level to which pregnant monkeys were exposed is approximately 10.000 times higher than typical human exposure.
The new study published in the Journal of the American Medical Association provides no evidence that BPA causes obesity. The authors of the study themselves state: ‘Obesity develops over time, and causation cannot be inferred from a cross-sectional association of urinary BPA concentration…’ (Read more)
A new scientific evaluation of the recent Vandenberg et al low dose study was conducted by Rhomberg/Goodman. The evaluation concluded that the Vandenberg et al study fails to provide sufficient evidence for low-dose effects and overlooks the evidence against low-dose effects.
The new Melzer et al. study lacks evidence to demonstrate BPA causes coronary artery disease. It is a small-scale study and it does not establish a cause-and-effect relationship between BPA exposure and coronary artery disease.
On 17 July, FDA decided to act on the American Chemistry Council (ACC) petition to revise the regulation of BPA in baby bottles and sippy cups.
The Swedish Food Agency noted that the amount of BPA entering the body of children through materials in contact with food is too small to pose any risk to children´s health.
The recent study by Soto et al is small in scale and did not test exposure to low doses of BPA, but to one single dose level which was 10,000 times higher than typical human exposure to BPA.
The US Food and Drug Administration (FDA) rejected a petition from the Natural Resources Defense Council (NRDC) to ban BPA in food-contact materials. This decision again confirms that BPA is safe for use in food-contact materials.
CHEM Trust and BUND published a report accusing BPA amongst several other chemicals to have effects on obesity and diabetes in humans. There is no scientific evidence of a link between BPA and obesity or diabetes. The literature research published by ChemTrust and BUND provides no new data on this topic.
The Melzer et al study is incapable of assessing or establishing a cause-effect relationship between BPA exposure and coronary artery disease. It is based on statistical models that cannot confirm such a link and it was performed on one single urine sample, giving no information on BPA exposure over the duration of the study (i.e. 10 years).